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  Indian J Med Microbiol
 

Figure 2: (a) Neuron-specific enolase (NSE) levels of two groups at analyzed time points. NSE levels were significantly higher in group propofol than group sevoflurane only at T5, (* P = 0.046). (b) NSE levels of group sevoflurane at analyzed time points. NSE levels at T3 were significantly higher compared to T1, T2, T5 and T6 (* P < 0.05 for all compares) and NSE levels at T4 were significantly higher than T1 (* * P = 0.047). (c) NSE levels of group propofol at analyzed time points. NSE levels at T3 were significantly higher compared to T1 and T2, (* P < 0.05 for all compares), NSE levels at T4 were significantly higher than T1 and T2 (* * P < 0.05) and NSE levels at T5 were significantly higher than T1 (* * * P < 0.05). Values are expressed as mean ± SD. T1 = After induction of anesthesia (baseline), T2 = 30 min of CPB, T3 = At the end of CPB, T4 = 1 h postoperatively, T5 = 6 hpostoperatively and T6 = 24 h postoperatively

Figure 2: (a) Neuron-specific enolase (NSE) levels of two groups at analyzed time points. NSE levels were significantly higher in group propofol than group sevoflurane only at T5, (* <i>P</i> = 0.046). (b) NSE levels of group sevoflurane at analyzed time points. NSE levels at T3 were significantly higher compared to T1, T2, T5 and T6 (* <i>P</i> < 0.05 for all compares) and NSE levels at T4 were significantly higher than T1 (* * <i>P</i> = 0.047). (c) NSE levels of group propofol at analyzed time points. NSE levels at T3 were significantly higher compared to T1 and T2, (* P < 0.05 for all compares), NSE levels at T4 were significantly higher than T1 and T2 (* * <i>P</i> < 0.05) and NSE levels at T5 were significantly higher than T1 (* * * <i>P</i> < 0.05). Values are expressed as mean ± SD. T1 = After induction of anesthesia (baseline), T2 = 30 min of CPB, T3 = At the end of CPB, T4 = 1 h postoperatively, T5 = 6 hpostoperatively and T6 = 24 h postoperatively